Neuropilin-1 is essential for vascular endothelial growth factor A–mediated increase of sensory neuron activity and development of pain-like behaviors

aDepartment of Molecular Pathobiology, College of Dentistry, New York University, New York, NY, United States

bNYU Pain Research Center, New York, NY, United States

cDepartment of Pharmacology, College of Medicine, The University of Arizona, Tucson, AZ, United States

dDepartment of Pharmacology and Physiology, School of Medicine, Saint Louis University, Saint Louis, MO, United States

eDepartment of Neuroscience and Physiology, New York University Grossman School of Medicine, New York, NY, United States

*Corresponding author. Address: Department of Molecular Pathobiology, College of Dentistry, New York University, 433 1st Ave Room 720, New York City, NY 10010, United States. E-mail address: [email protected] (R. Khanna).

Sponsorships or competing interests that may be relevant to content are disclosed at the end of this article.

Supplemental digital content is available for this article. Direct URL citations appear in the printed text and are provided in the HTML and PDF versions of this article on the journal's Web site (www.painjournalonline.com).

K. Gomez and P. Duran are co-first authors.

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