Gram-positive anaerobic cocci (GPAC) are a significant component of the normal human commensal flora, residing on the skin and mucosal surfaces of the oral cavity, as well as in the respiratory, gastrointestinal and genitourinary tracts [1]. They account for 25–30 % of all anaerobes recovered from human clinical specimens [1].

Among GPAC, Finegoldia magna (formely known as Peptostreptococcus magnus) is the most commonly isolated species in human pathology, particularly in polymicrobial infections such as skin and soft tissue infections, bone and joint infections, and diabetic foot ulcers [[1], [2], [3]]. The most frequent infections involving Finegoldia magna as the sole pathogen are septic arthritis, osteomyelitis and soft-tissue infection, particularly in the presence of prosthetic devices or materials [[2], [3], [4], [5]].

Finegoldia magna is a rare and unusual pathogen in infective endocarditis (IE). The first documented case of F. magna as a causative agent of IE occurred in 1985, involving a native mitral valve infection [6]. Since then, only eleven additional cases of IE caused by this bacterium have been reported in the medical literature, one of which involved a native mitral valve [7], with the remaining ten involving prosthetic valves [[8], [9], [10], [11], [12], [13], [14], [15]]. Here, we report a new case of infective endocarditis on a prosthetic valve caused by Finegoldia magna, along with comprehensive literature review of Finegoldia magna infective endocarditis.